Editorial: Genetics of Brain Function and Cognition
نویسندگان
چکیده
489 0001-8244/01/1100-0489$19.50/0 © 2001 Plenum Publishing Corporation If there is any truth in the allegation that behavior geneticists like behaviors best when they show high heritability, then surely filling out an IQ test is the human behavior that makes us most happy. Extensive research based on twin, family, and adoption data has documented that more than half of the striking individual differences in adult IQ test performance are due to genetic factors (Bouchard and McGue, 1981; Boomsma, 1993; Devlin et al., 1997; McClearn et al., 1997; Plomin, 1999; Wright et al., 2001). This finding applies across many countries and across the entire adult age range. In spite of the overwhelming evidence for the existence of “genes of cognitive ability,” actual identification of such genes is limited to neurological mutations with rather severe cognitive effects (e.g., Pick’s disease, X-linked mental retardation, Huntington’s disease) as reviewed by Flint (1999). Like the many rare diseases and disorders listed in Online Mendelian Inheritance in Man (OMIM) (McKusick, 1998) these genetic defects of cognition are largely Mendelian in nature. True polygenes (or QTLs) that influence the normal range of cognitive ability have Editorial
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